tetano
Editor, Senior Moderator
J Infect Dis. 2017 Sep 15;216(6):697-702. doi: 10.1093/infdis/jix393.
[h=1]Age-Related Differences in Influenza B Infection by Lineage in a Community-Based Sentinel System, 2010-2011 to 2015-2016, Canada.[/h] Skowronski DM[SUP]1,[/SUP][SUP]2[/SUP], Chambers C[SUP]1[/SUP], De Serres G[SUP]3,[/SUP][SUP]4,[/SUP][SUP]5[/SUP], Sabaiduc S[SUP]1[/SUP], Winter AL[SUP]6[/SUP], Dickinson JA[SUP]7[/SUP], Gubbay JB[SUP]6,[/SUP][SUP]8[/SUP], Fonseca K[SUP]7,[/SUP][SUP]9[/SUP], Drews SJ[SUP]10,[/SUP][SUP]11[/SUP], Charest H[SUP]3[/SUP], Martineau C[SUP]3[/SUP], Krajden M[SUP]1,[/SUP][SUP]2[/SUP], Petric M[SUP]2[/SUP], Bastien N[SUP]12[/SUP], Li Y[SUP]12[/SUP].
[h=3]Author information[/h]
[h=3]Abstract[/h] Age-related differences in influenza B lineage detection were explored in the community-based Canadian Sentinel Practitioner Surveillance Network (SPSN) from 2010-2011 to 2015-2016. Whereas >80% of B(Victoria) cases were <40 years old, B(Yamagata) cases showed a bimodal age distribution with 27% who were <20 years old and 61% who were 30-64 years old, but with a notable gap in cases between 20 and 29 years old (4%). Overall, the median age was 20 years lower for B(Victoria) vs B(Yamagata) cases (20 vs 40 years; P < .01). Additional phylodynamic and immuno-epidemiological research is needed to understand age-related variation in influenza B risk by lineage, with potential implications for prevention and control across the lifespan.
? The Author 2017. Published by Oxford University Press for the Infectious Diseases Society of America.
[h=4]KEYWORDS:[/h] age; birth cohort effects; influenza B lineage; influenza B virus; risk
PMID: 28934439 DOI: 10.1093/infdis/jix393
[h=1]Age-Related Differences in Influenza B Infection by Lineage in a Community-Based Sentinel System, 2010-2011 to 2015-2016, Canada.[/h] Skowronski DM[SUP]1,[/SUP][SUP]2[/SUP], Chambers C[SUP]1[/SUP], De Serres G[SUP]3,[/SUP][SUP]4,[/SUP][SUP]5[/SUP], Sabaiduc S[SUP]1[/SUP], Winter AL[SUP]6[/SUP], Dickinson JA[SUP]7[/SUP], Gubbay JB[SUP]6,[/SUP][SUP]8[/SUP], Fonseca K[SUP]7,[/SUP][SUP]9[/SUP], Drews SJ[SUP]10,[/SUP][SUP]11[/SUP], Charest H[SUP]3[/SUP], Martineau C[SUP]3[/SUP], Krajden M[SUP]1,[/SUP][SUP]2[/SUP], Petric M[SUP]2[/SUP], Bastien N[SUP]12[/SUP], Li Y[SUP]12[/SUP].
[h=3]Author information[/h]
[h=3]Abstract[/h] Age-related differences in influenza B lineage detection were explored in the community-based Canadian Sentinel Practitioner Surveillance Network (SPSN) from 2010-2011 to 2015-2016. Whereas >80% of B(Victoria) cases were <40 years old, B(Yamagata) cases showed a bimodal age distribution with 27% who were <20 years old and 61% who were 30-64 years old, but with a notable gap in cases between 20 and 29 years old (4%). Overall, the median age was 20 years lower for B(Victoria) vs B(Yamagata) cases (20 vs 40 years; P < .01). Additional phylodynamic and immuno-epidemiological research is needed to understand age-related variation in influenza B risk by lineage, with potential implications for prevention and control across the lifespan.
? The Author 2017. Published by Oxford University Press for the Infectious Diseases Society of America.
[h=4]KEYWORDS:[/h] age; birth cohort effects; influenza B lineage; influenza B virus; risk
PMID: 28934439 DOI: 10.1093/infdis/jix393